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Oxidative inactivation of key mitochondrial proteins leads to dysfunction and injury in hepatic ischemia reperfusion

CYP7A1 is simultaneously inhibited by an FXR-small heterodimer partner (SHP), which is the downstream target of FXR in the liver, whereas FXR in enterocytes inhibits CYP7A1 activation in hepatocytes by secreting fibroblast growth factor 19 (FGF19) and binding to fibroblast growth factor receptor 4 (FGFR4) on the hepatocyte membrane (77)