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Description

Unlike GLP-1 agonists that reduce appetite or growth hormone peptides that stimulate lipolysis, the compound addresses fat metabolism at the enzymatic level: it removes the molecular governor that locks adipocytes into a low-energy-expenditure state, potentially restoring normal metabolic function in fat tissue where NNMT is overexpressed

During the acute phase, MP infection also induces IL-23 production by alveolar macrophages

In agreement with a role of HMOX1 as a driver of immunosuppression, myeloid-specific HMOX1 deletion restored CD8 + T cell function and enhanced the therapeutic efficacy of antitumor vaccines [80]