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Notably, a discrepancy exists between the proportion of cells undergoing classical apoptosis and the overall reduction in cell viability measured by the CCK-8 assay (Figures 1A,B, 3A,B)

From a mechanistic perspective, peroxisomal -oxidation dysfunction in AP would be expected to exert a dual detrimental effect: first, by impairing the shortening of fatty acids before their transfer to mitochondria, and second, by weakening peroxisomal control of hydrogen peroxide and related redox signals

K., Jbara A., Rabaya O., Petrova P., Daoud S., Melliti N., et al

Grama CN, Suryanarayana P, Patil MA, Raghu G, Balakrishna N, Ravi Kumar MNV, et al