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In those models, disturbed iodine availability impairs the synthesis of thyroid hormones, resulting in compensatory enlargement of the thyroid

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Activation of tetrodotoxin-resistant sodium channel naV1.9 in rat primary sensory neurons contributes to melittin-induced pain behavior

[1,21] The exceptions identified in the studies mentioned in the previous paragraph suggest that a massive overdose may produce a mismatch between N-acetyl-p-benzoquinone imine (NAPQI) (the toxic metabolite of acetaminophen) formation and the detoxification by conjugation to glutathione