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Further studies have shown that due to heme-induced mitochondrial oxidative stress, the present ALAS2 transgenic mice has exhibited an increase in heme accumulation, aggravation of cell death, and exacerbation of cardiac dysfunction after coronary ligation compared with the control littermates (Sawicki et al., 2015b)

While not entirely comprehensive and lacking robust evidence, this hypothesis has spurred significant research in the field

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The following table summarizes common claims and their evidence basis: The key pattern: most benefits are real only for individuals who are actually deficient in B12