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The details of such reactions are extensively reviewed in previous publications [77, 79]

A combination of low selenium and genetics may play a role in the susceptibility to Keshan disease as individuals carrying the Leu198 variant of GPX1 , which is associated with lower expression, especially in the context of lower selenium levels, are at greater risk of developing Keshan disease [36]

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The key effects are well-established: accelerated cell turnover (the outer layers of the stratum corneum, the skin's surface barrier, shed more quickly), direct stimulation of procollagen genes, and suppression of matrix metalloproteinases - the enzymes responsible for breaking down existing collagen