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Gln inhibits miR-29b-3p transcription via c-Myc regulation to promote GGCT protein expression To determine GGCT downregulation during Gln deprivation, we first excluded protein degradation pathways as proteasomes (MG132) or lysosomes (CQ) inhibition failed to restore GGCT levels (Fig

Since this appeared to be a global trend, we hypothesized that circulating triglycerides were depleted in Gclc KO mice

By exponentially increasing the production of Glutathione S-transferases (GSTs) and supplying necessary sulfur for the sulfation pathway, NAC ensures that the liver can safely conjugate and excrete toxic intermediate metabolites without causing secondary tissue damage