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However, the mechanisms by which selenium metabolism regulates chondrogenic programs remain still unclear and require further investigation

10.1016/j.gie.2017.05.036 13 AlonsoJ.PetukhovaM.VilagutG.ChatterjiS.HeeringaS.stnT

Healing rates, loading patterns, and the architecture of the tendon-bone interface differ

Under nutrient-abundant conditions, it phosphorylates ULK1 to suppress mitophagy, whereas under nutrient-limited conditions, mTORC1 is inactivated, allowing AMPK to activate ULK1 by phosphorylation, thereby inducing mitophagy (97)