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parenterally or perorally) consistently antagonizes the NSAIDs effect on lower esophageal sphincter and pyloric sphincter, thus counteracting both the early effects and the late effects induced by non-specific and specific NSAIDs

The assumption that high-dose synthetic BPC-157 injection has the same effects as the endogenous gastric compound is not yet proven

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Additionally, ROS activates stress-sensitive kinases (JNK, p38 MAPK) that phosphorylate IRS-2, accelerating its proteasomal degradation and further impairing -cell survival (Liu et al., 2020)