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doi: 10.1210/en.2006-1285

Because it enhances detox enzyme activity, it may either reduce medication effectiveness or change how quickly drugs are cleared from the body

As a result, laboratories can maintain confidence in the consistency of the materials they evaluate

Given that RIPK1 is also integral to the progression of these diseases, we propose the hypothesis that HK2 may regulate RIPK1 via its non-metabolic functions, thereby influencing the progression of cell death