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Emerging evidence further indicates that MTs particularly MT1 and MT2A constitute a critical cytoplasmic defense layer against cuproptosis by acting as effective copper sponges that sequester excess Cu + and restrict its aberrant interaction with mitochondrial targets 170,173,174

ROS then interacts with lipids in the cell membranes causing cellular death

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3 in which the rate of ASD continued to climb as direct-to-consumer advertising in the US increased dramatically and then became a part of US culture