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Lack of SMARCA4 inhibits expression of the aldehyde dehydrogenase ALDH16A1 that binds to the Trx1 active site and inhibits its function but also promotes lysosomal degradation of the redoxin [95]

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Inflammatory mediators such as cytokines, chemokines, and adhesion molecules are upregulated in HF, contributing to the progression of the disease [3]