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Inhibiting CPT1A causes cells to deplete stored aspartic acid and dNTP, impairs de novo dNTP synthesis, and inhibits NPC cell cycles DNA replication at G1/S transition, implying a potential treatment strategy for NPC based on lipid metabolism regulation (Schoors et al., 2015

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Biomarkers for endothelial function, insulin sensitivity, intracellular signaling, maintaining DNA integrity , inflammation, metabolic flexibility, mitophagy (recycling old and damaged mitochondria) and cellular senescence (also called "zombie cells" as these cells have lost proper function, harm nearby cells and do not die off) are all favorably affected