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This section delves into the mechanistic pathways through which AS-IV modulates these critical pathological processes (Figure 1)

two daughters, Rachel Lauren Nease and Sarah Catherine Cottam, both of Jonesboro

The COLBOS mutation was linked to stronger activation of the canonical reelin target Dab1, and this signaling pathway was shown to directly lead to reduction in tau phosphorylation and amelioration of the phenotype in a mouse model of tauopathy [197]