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Pathological iron accumulation appears to result from altered expression and function of iron transport proteins at the blood-brain barrier, exacerbated by inflammatory cytokines and oxidative stress that modulate key iron transporters such as transferrin receptor, divalent metal transporter 1, and ferroportin

Conclusion By integrating bioinformatics prediction with in vivo validation, this study elucidates that palmatine alleviates T2DM-associated MASLD by targeting a network of five key proteins (ADRB2, BCL3, EGR1, FOS, MAP3K8)

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Product quality also emerges as a consistent factor