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Researchers propose that the peptide's brief presence is sufficient to trigger a cascade of gene expression changesincluding activation of transcription factors c-Fos, c-Jun, and Egr-1that initiate self-sustaining healing processes well beyond the peptide's physical presence

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Tendon collagen turns over slowly, the blood supply to tendons is inherently limited, and the mechanical demands on tendons mean they need to rebuild strength progressively rather than all at once