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Technically, but it wasn't designed for that purpose

The possibility that sustained folic acid intake would deplete vitamin B-12 independent of hematopoiesis suggests a plausible alternative mechanism based on the known details of the renal handling of holoTC, whereby folic acid might interfere with holoTC reuptake or cause its sequestration in the kidney, promoting the excretion of holoTC and further depleting metabolically active vitamin B-12 during vitamin B-12 deficiency

The structural rearrangements of the host cells will likely be highly beneficial for the pathogen

That approach prevents the article from using selective literature snippets as marketing language