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Please refer to the full prescribing information for pembrolizumab and for carboplatin or cisplatin

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There is a mounting body of evidence indicating a close association between cellular senescence and the underlying pathophysiology of AKI, as they both exhibit a cell cycle arrest at the G2/M phase, heightened oxidative stress, an increase in cyclin-dependent kinase inhibitors, telomere shortening, and an exacerbated in the fibrotic processes

Silencing of either SR-A or CD36 reduces atherosclerosis in hyperlipidaemic mice and reveals reciprocal upregulation of these receptors