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Enhanced fibroblastic activity increases tissue-healing processes of chemotaxis, proteosynthesis, reparation, extracellular matrix deposition, and tissue remodeling

Late in treatment cycles and towards end of treatment (EOT), the CTCs in both patients showed decreases in BCL-2 and AR expression, AR activity ( KLK3 and TMPRSS2 ), and overall abundance as evidenced by reduced EPCAM and the TMPRSS2 -ERG fusion (supplementary Fig