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We therefore tested the role of neuronal activity-regulated BDNF secretion into the tumour microenvironment using a genetically engineered mouse model that is deficient in activity-induced expression of BDNF 32 ( Bdnf- TMKI)

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These findings suggest that information from cells in which CaSR agonists have bound to the receptors might not be sent to the brain, but may instead be used to modify other taste responses within taste buds