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Most notably, CO, the end product of HO-1 activity, has been suggested as a therapeutic mimic of HO-1, either through gas administration or pharmacological delivery of chemical donor compounds, including transition metals, CO-releasing molecules (CORMs), and organic CO donors containing carbon monoxide-releasing molecules (Steiger et al., 2017

282 This treatment dilemma is actually exacerbated by the increasing demands on LDL levels in patients with cardiovascular disease

Schmit PJ, Hiyama DT, Swisher SG , Thompson JE

The dynamic exclusion time was set to 20 s and the peptide match and isotope exclusion functions were enabled