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In laboratory research, glutathione is extensively studied for its roles in phase II xenobiotic metabolism, where it conjugates with reactive electrophiles via glutathione S-transferase enzymes

They show that the presence of hydroquinone alone is not enough to inhibit microbial cell growth significantly and that benzoquinone shows more potential as an antimicrobial compound with possible pharmaceutical use

These findings may reflect an immediate stress response following ECT [38], as electrophysiological intervention might temporarily promote the conversion of GSH to GSSG [39]