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Copper toxicosis, due to failure to transport this metal into the biliary canaliculus, is the mechanism of liver damage in Wilson disease, a genetic disease caused by a loss-of-function mutation in the ATP7B gene

Wheat amylase trypsin inhibitors drive intestinal inflammation via activation of toll-like receptor 4

Functional improvements generally become noticeable during weeks 2 to 4

Further studies are needed to determine how the glutathionylation of RBP proteins occurs during the progression of ALS and how it is linked to the pathogenesis of ALS