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The brain on drugs: from reward to addiction

The two forms of autophagy-related cell death are not independent of each other and can be triggered simultaneously, and cell death processes may switch between these two modalities under certain conditions [26]

At the immune level, metabolic crosstalk critically shapes polarization: persistent long-chain fatty acid (LCFA) exposure depletes intestinal SCFAs while promoting differentiation of nave T cells into pro-inflammatory Th1/Th17 subsets in the small intestine (Haghikia et al., 2015)

found that animals with multiple sclerosis had significantly lower levels of catalase in the hippocampus, but no changes were observed in plasma