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Alterations in aerobic metabolism have been reported in fibroblasts involved in pulmonary fibrosis [166], particularly increased glycolysis, resistance to apoptosis, and propensity to develop a metabolic-driven fibrotic phenotype, which appears to be linked to decreased ATP synthesis and increased mtROS production [169, 170]

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We constructed bisulfite sequencing libraries for three replicates of different tissues, and conducted genome sequencing using the Illumina HiSeq X platform 95