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Furthermore, a study conducted on human retinal cells (ARPE-19) exposed to sources of oxidative stress, such as hydrogen peroxide (HO) or UVB rays, demonstrated that each molecule has a distinct antioxidant effect

In this manner, mass action and/or altered mitochondrial redox states induce proliferating cells to reductively metabolize aKG via NADP + -dependent IDHs and subsequently generate isocitrate and ultimately AcCoA [49]