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Description

It is a critical component in studies involving Phase II detoxification pathways and the protection of cellular macromolecules from oxidative damage

Given the critical role of neuroinflammation in AD pathology, further research using Lewiss rat models could uncover YSTJs efficacy in modulating microglial activity, reducing pro-inflammatory cytokines, and preserving neuronal integrity

This also allows for higher dosages to be used safely compared to oral supplementation

While human clinical trials remain limited, animal studies and extensive anecdotal evidence provide guidance for researchers and those exploring this peptide