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EEL effectively modulates the TGF/Smads pathway, which links oxidative stress to inflammatory responses and tissue remodeling, by reducing the expression of TGF2 and Smad2 while increasing inhibitory Smad7 [248]

[14] [15] The administration of N-acetylcysteine (NAC), a cysteine prodrug, helps replenish intracellular GSH levels

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