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This may be explained by the changing metabolic state of injured cells or organs during the pathological process of disease

Sodium MRI in multiple sclerosis is compatible with intracellular sodium accumulation and inflammation-induced hyper-cellularity of acute brain lesions

The first category involves metabolites serving as essential nutrients, energy sources, and building blocks required for the development, differentiation, proliferation, and functional activity of immune cells

Enhancing FAO via LCA supplementation (the cofactor of CPT1B), attenuated cardiac steatosis and lipotoxicity-induced pathological changes in the atria of obese mice, resulting in restored AF substrates and ameliorated AF susceptibility (Figure 7)