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FEMS Microbiol Rev 45:fuaa061 Kramnik I, Beamer G (2016) Mouse models of human TB pathology: roles in the analysis of necrosis and the development of host-directed therapies

A frame-shift null mutation (c.523delC, p.Q175RfsX38) of SLC25A4 resulted in autosomal recessive myopathy and cardiomyopathy

The same study also found that NAC protected ovalbumin-sensitized mice against DEP exposure, although vitamin C and E were not examined in this experiment