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10.7150/thno.21389 105 JiangL.KonN.LiT.WangS.-J.SuT.HibshooshH.et al (2015)

Driessen, R

These results suggested that oxidative stress-mediated cellular senescence 18 might be responsible for the observed phenotype

The alteration of PDI/UPR pathway in neurodegenerative diseases has been nicely reviewed in [63] but the following highlights from brain post-mortem samples should be considered