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Using in vivo studies, they demonstrated that a B vitamin deprivation diet led to hyperhomocysteinemia, an imbalance of SAM/SAH, PS1, and BACE upregulation, each of which increased A deposition in TgCRND8 mice [142, 143]

Notably, the data showed that MUC1 high-ATAD3A low patients ( n = 228) were considerably associated with shorter overall survival compared to MUC1 low-ATAD3A high patients ( n = 263) (Fig

By regulating these molecules and restoring disrupted pathways, TQ may play a valuable role in modulating immune responses and inflammatory reactions associated with autoimmune dysfunction (80, 84)

XBP-1 deficiency in the nervous system protects against amyotrophic lateral sclerosis by increasing autophagy