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Consequently, disruption of mitochondrial transport to the synapse is a primary driver of synaptic degeneration and eventual neuronal death ( Evidence from cellular and murine models indicates that Tau overexpression and hyperphosphorylation severely disrupt the localization and distribution of mitochondria ( Similarly, iPSC-derived neurons with the N279K and P301L Tau mutations show a significant reduction in anterograde axonal transport compared to healthy controls ( 5.3 ROS-driven amplification loops Mitochondrial dysfunction is an early-stage hallmark of AD, characterized by diminished ATP synthesis and a reciprocal surge in reactive oxygen species (ROS) (89)

Correction of anemia, hyperparathyroidism, and 1, 25-OH vitamin D3 deficiency and provision of adequate dialysis do not fully restore muscle function and exercise capacity in ESRD patients

We tried to measure the changes of the global level of sleepiness using total dozing off time on sleep logs and JESS rating