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Taken collectively, our data suggest that metformin treatment expanded the abundance of ETC-related proteins that, upon treatment cessation at D3, allowed for OXPHOS to occur in activated B cells with minimal mitochondrial oxidative stress

The distributions show fold-changes (log 2 ) in miRNA levels in Zswim8 -knockout mouse embryonic fibroblasts (MEFs), relative to control cells ( n = 3 biological replicates)