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The pathogenesis of POAG can be summarized as follows: endoplasmic reticulum (ER) stress promotes the excessive deposition of ECM in the trabecular meshwork (TM) by interfering with ECM protein processing, thereby inducing TM fibrosis

- Niacin (Vitamin B3) 3%

BPC-157 sits in the pre-clinical stage for reproductive health, though its anti-inflammatory and gut-protective effects are relevant to the PMOS disease profile

Thijssen MA, Hornstra G, Mensink RP (2005) Stearic, oleic, and linoleic acids have comparable effects on markers of thrombotic tendency in healthy human subjects