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10.1186/s13041-015-0116-z Mol

Neuroprotection: Preclinical research has explored possible protective effects in age-related neurological studies

The tissue-repair mechanisms are highly relevant to these goals in a way NAD+ is not

Using the Broad Institutes Connectivity Map dataset, Pickart and colleagues reported that GHK significantly changed the expression of a very large number of human genes (on the order of thousands when a modest expression-change threshold is applied), up-regulating many and down-regulating others in a pattern the authors interpret as a shift toward a healthier, more repair-competent state