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4.2 - GVL mechanisms of DLI Relapse of hematologic malignancy after HSCT may be at least in part attributable to development of T cell dysfunction due to exhaustion and therefore loss of the protective GVL effect endowed by HSCT

non-progressive disease

When there is a quantitative deficiency in the alpha or beta chains, membrane-bound iron becomes free and can react with reduced glutathione, as we have shown

It is necessary to acknowledge that this mechanism is totally distinct from the previous observation of the preventive effect of LASSS on HGF nitration (1:4000 ratio, without the wash-out of un-reacted LASSS