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and Nova Southeastern University for funding our research

found that I/R was able to lead to liver damage, LPO as well as upregulation of the ferroptosis marker Ptgs2 in a murine model, and all of these effects were significantly alleviated by ferroptosis-specific inhibitor ferrostatin-1 and iron chelator deferoxamine [18]

Thus, in a physiological context inflammation is protective and once the insult is eradicated several mechanisms intervene and lead to a process named resolution of inflammation

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