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Finally, add DAPI-containing anti-fluorescence quenching mounting medium and mount the slides

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Taken together, these preclinical data support interventions that target UA to counteract oxidative stress in the ischemic penumbra and protect endothelial cell function after cerebral ischemia-reperfusion injury

Key questions remain regarding its ability to effectively inhibit NLRP3 inflammasome activation within the complex milieu of diabetic lung injury, and whether long-term suppression of this core innate immune pathway might lead to the activation of compensatory inflammatory pathways or other unknown off-target effects, all of which require longer-term clinical investigation