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Histopathological examination of major organs including liver, kidney, heart, brain, and reproductive tissues shows no treatment-related lesions

These include genetic, oxidative stress, autoimmune, autoinflammatory, neural, apoptotic, molecular adhesion disorder and multifactorial mechanisms [1]

However, these findings were not validated in animal models, and key pharmacological parametersincluding fertility outcomes, long-term toxicity, and pharmacokineticsremain uncharacterized

In previous studies, cellular senescence has been considered a mechanism of preventing abnormal proliferation to malignancy in keloids and could be used to halt the progression of keloids 13